Attenuation of Tau-Mediated β-Amyloid1-42 Aggregation by Native PLGA Nanoparticles and Its Relevance to Alzheimer Disease Pathology.
International journal of molecular sciences · 2026
Abstract e pubblicazione originale
L’abstract è consultabile nella fonte originale. Non viene ripubblicato qui perché non risulta verificata una licenza compatibile.
Leggi nella fonte originale ↗Parole chiave
Amyloid beta-Peptidestau ProteinsAlzheimer DiseaseNanoparticlesPeptide FragmentsPolylactic Acid-Polyglycolic Acid CopolymerHumansProtein AggregatesAnimalsProtein Aggregation, PathologicalAlzheimer’s diseaseAβ aggregationPLGA nanoparticlestau monomertau seeds
Come citare questo studio
Pallabi Sil Paul, Aliza Borenstein-Katz, Klinton Shmeit, Holger Wille, Satyabrata Kar. (2026). Attenuation of Tau-Mediated β-Amyloid1-42 Aggregation by Native PLGA Nanoparticles and Its Relevance to Alzheimer Disease Pathology.. International journal of molecular sciences. DOI: 10.3390/ijms27188152
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