Alda-1 alleviates the cognitive deficits in D-galactose-induced aging mice by regulating oxidative stress, neuroinflammation, and mitochondrial biogenesis.
The journals of gerontology. Series A, Biological sciences and medical sciences · 2026
Abstract e pubblicazione originale
L’abstract è consultabile nella fonte originale. Non viene ripubblicato qui perché non risulta verificata una licenza compatibile.
Leggi nella fonte originale ↗Parole chiave
AnimalsOxidative StressGalactoseMiceOrganelle BiogenesisAgingCognitive DysfunctionBenzamidesMaleBenzodioxolesHippocampusNeuroinflammatory DiseasesAldehyde Dehydrogenase, MitochondrialMitochondriaMice, Inbred C57BLPeroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alphaALDH2Cognitive deficitsMitochondrial biogenesisOxidative stress
Come citare questo studio
Tengfei Liang, Lixia Wang, Shujuan Kang, Xiaoyan Pang, Hexin Yan. (2026). Alda-1 alleviates the cognitive deficits in D-galactose-induced aging mice by regulating oxidative stress, neuroinflammation, and mitochondrial biogenesis.. The journals of gerontology. Series A, Biological sciences and medical sciences. DOI: 10.1093/gerona/glag241
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