40 Hz vibrotactile stimulation ameliorates amyloid pathology and cognitive deficits in 5xFAD mice and is associated with alterations in Piezo1, ERK, and GSK-3β/p65 signaling.
Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics · 2026
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AnimalsGlycogen Synthase Kinase 3 betaAlzheimer DiseaseMiceMice, TransgenicAmyloid beta-PeptidesSignal TransductionIon ChannelsVibrationCognitive DysfunctionCognitive EnhancementMalePhysical StimulationMAP Kinase Signaling SystemAlzheimer'sAmyloid betaERK pathwayGSK-3βPiezo1Vibrotactile stimulation
Come citare questo studio
Myeong-Hyun Nam, Hee-Jung Park, Hee-Yeon Lee, Zu-Yu Chen, Hee-Deok Yun, Yuna Kim, Ju-Yeong Lee, Chang-Ho Shin, Jae-Young Ha, Young-Kwon Seo. (2026). 40 Hz vibrotactile stimulation ameliorates amyloid pathology and cognitive deficits in 5xFAD mice and is associated with alterations in Piezo1, ERK, and GSK-3β/p65 signaling.. Neurotherapeutics : the journal of the American Society for Experimental NeuroTherapeutics. DOI: 10.1016/j.neurot.2026.e01077
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